This study assessed the effect of providing free malaria treatment in the Buea health district.
In this study, we show that children with SMA have elevated levels of circulating sLAIR1 and sLAIR2, indicative of enhanced receptor shedding.
In this review, we concentrate on the role of glycophorins as erythrocyte receptors for Plasmodium parasites.
This study aims to determine whether cochlear malfunction persists for 4 years after recovery from severe malaria in a subset of the previous study's collective.
Here, a large cohort of vivax malaria and HBV patients is retrospectively analyzed in an attempt to depict how inflammatory characteristics could be potentially related to the protection to severe malaria in coinfection.
We explored 3 immunopathogenic biomarkers collected during acute malaria illness as potential moderators of gains from a computerized cognitive rehabilitation training (CCRT) intervention.
In this review, researchers from The Cochrane Collaboration examined the effects of treating people that have severe malaria with artemether injected intramuscularly, and compared it to treatment with other antimalarial drugs given intramuscularly or intravenously.
Here we present an approach to estimate the latent determinants of parasite load dynamics. We use these estimates to better understand severe malaria phenotypes and to identify mechanisms inhibiting parasite growth and controlling parasite multiplication during Plasmodium falciparum malaria in Gambian children.
This retrospective study included 75 cases diagnosed as having imported malaria in our clinic between January 2008 and December 2017
To clinically and epidemiologically characterize malaria episodes in hospitalized pregnant women in the Department of Antioquia (Colombia) in the period 2010-2014
We aimed to determine if delaying iron until 28 days after antimalarial treatment in children with severe malaria and iron deficiency leads to fewer subsequent clinical malaria episodes as compared to concurrent iron therapy.
Here, we establish that unlike P. falciparum, P. knowlesi and P. vivax do not universally require BSG as a host cell invasion receptor.